NCC Certified - Electronic Fetal Monitoring EFM Exam Dumps: Updated Questions & Answers (November 2025)

Comprehensive and Detailed Explanation From Exact Extract Without Links:

NCC relies heavily on ACOG Practice Bulletins for risk-based monitoring decisions. ACOG identifies maternal diabetes (pregestational or poorly controlled gestational diabetes) as a key high-risk obstetric condition warranting continuous electronic fetal monitoring due to risks such as fetal hypoxia, macrosomia, and metabolic complications.

In contrast, a history of preterm birth does not necessarily require continuous monitoring unless current pregnancy complications are present.

Macrosomia alone does not automatically justify continuous EFM unless accompanied by other risk factors.

Therefore, according to NCC-aligned ACOG clinical criteria, maternal diabetes is the correct indication.

Comprehensive and Detailed Explanation From Exact Extract–Based NCC C-EFM References:

This tracing demonstrates:

A sudden prolonged deceleration following epidural placement

Minimal variability during the deceleration

Event occurring within 15 minutes of epidural

NCC, AWHONN, and Menihan emphasize that maternal hypotension is the most common complication immediately following epidural analgesia. Hypotension leads to:

Reduced uteroplacental perfusion

Fetal bradycardia or prolonged decelerations

Decreased variability during the deceleration

Typical fetal response to maternal hypotension:

Late-like or prolonged deceleration with weakening variability, exactly like the strip shown.

Therefore, the FIRST and most critical step is to check maternal blood pressure.

Other options:

B. Continue to monitor — unsafe when a prolonged deceleration is present.

C. Cervical exam — not indicated; the fetal tracing deterioration is temporally linked to epidural placement.

Thus, the correct action is A. Check maternal blood pressure.

Comprehensive and Detailed Explanation From NCC-Aligned Sources:

Vagal stimulation is part of the parasympathetic nervous system, which causes:

Slowing of the fetal heart rate (FHR)

Rapid but temporary changes in HR

Seen with head compression, scalp stimulation, or fetal movement

NICHD/NCC physiology explains:

Vagus nerve activation → acetylcholine release → slowed SA node firing → decrease in FHR

This mechanism is responsible for early decelerations during labor due to head compression.

Why the incorrect answers are wrong:

B. Increased cardiac contractility → sympathetic effect, not vagal.

C. Increased fetal blood pressure → also a sympathetic effect.

Correct answer: A. Decreased fetal heart rate

Comprehensive and Detailed Explanation From Exact Extract–Based NCC C-EFM References:

NCC and AWHONN physiology teachings:

Variable decelerations caused by cord compression lead to:

Transient interruption of umbilical venous flow

Impaired fetal gas exchange

Acute rise in CO₂

Respiratory acidosis (early phase of hypoxemia)

This is well documented:

Early decelerations → head compression → NOT associated with acidemia.

Late decelerations → uteroplacental insufficiency → metabolic acidosis, not respiratory.

Thus:

Variable decelerations → respiratory acidosis

Late decelerations → metabolic acidosis

Correct answer: C. Variable

Comprehensive and Detailed Explanation From Exact Extract–Based NCC C-EFM References:

The fetal heart rate tracing shows:

Baseline around 140 bpm

Minimal variability

No accelerations

No decelerations

Regular uterine activity but not tachysystole

This pattern is Category II, but in the context of:

28-week gestation

Immediate postoperative status after anesthesia

Maternal alertness and stability

NCC and AWHONN emphasize that maternal sedation, post-anesthesia effects, medications, and physiologic stress commonly cause temporary minimal variability without acidemia, especially at preterm gestations where baseline variability is normally lower.

Key NCC principle:

Minimal variability in a stable mother without decelerations does NOT require emergent delivery.

Instead, the fetus should be observed as anesthesia effects wear off.

Why other answers are incorrect:

A. Terbutaline – No tachysystole and no recurrent decels are present.

C. Cesarean birth – No bradycardia, no late decels, no absent variability, and no Category III criteria.

Thus, appropriate management is B. Continued monitoring.

Comprehensive and Detailed Explanation From Exact Extract–Based NCC C-EFM References:

This tracing demonstrates a normal, reassuring fetal heart pattern that is technically categorized as Category I, indicating normal fetal acid–base status. Before any decision regarding discharge or induction, NCC emphasizes correct assessment of the tracing quality, fetal status, and uterine activity.

Key Tracing Characteristics

Baseline:Approximately 135–145 bpm, well within the normal range of 110–160 bpm.

Variability:The strip shows moderate variability (6–25 bpm), the strongest indicator of adequate fetal oxygenation per NCC, AWHONN, and NICHD.

Accelerations:Several accelerations are present—another reassuring feature of normal fetal well-being.

Decelerations:No variable, late, or prolonged decelerations are present.

Uterine Activity:The lower channel shows poor recording quality and inconsistent signal—suggesting the toco is not capturing contractions well, not that the patient is contracting excessively or not at all.

Correct interpretation per NCC:

NCC emphasizes distinguishing between physiologic assessment and technical artifact.

The fetal tracing is completely reassuring.

The only abnormality is the poor uterine activity signal, a common triage occurrence due to:

Toco placement

Maternal body habitus

Positioning

Low contraction intensity in early labor

Thus, the correct next step is to optimize equipment (reposition the toco, adjust belt, palpate contractions) and continue to monitor.

Why the other options are incorrect:

B. Admit for induction – NOT indicated

There is no evidence of fetal compromise.

No indication for induction is present (pain score 4/10, reassuring FHR, term pregnancy).

NCC emphasizes avoiding unnecessary interventions.

C. Discharge to home – NOT yet appropriate

You cannot safely discharge a patient with a poorly monitored contraction pattern.

Adequate assessment requires confirming uterine activity—after fixing the toco.

Therefore, the appropriate action is:

A. Adjust tocotransducer and continue to monitor.

Comprehensive and Detailed Explanation From NCC-Aligned Sources:

Renal agenesis → severe oligohydramnios (due to absent fetal urine production).

Oligohydramnios causes:

Cord compression

Recurrent variable decelerations

Possible prolonged decels from cord entrapment

This is one of the hallmark FHR complications in renal agenesis.

Why the other options are incorrect:

A. Heart block — associated with maternal autoimmune antibodies, not renal anomalies.

B. Late decelerations — associated with uteroplacental insufficiency, not fluid deficiency.

Correct answer: C. Variable decelerations.

Comprehensive and Detailed Explanation From NCC-Aligned Sources:

NCC defines amnioinfusion as indicated for:

Recurrent variable decelerations caused by cord compression

Oligohydramnios reducing buffer around the cord

Expected effect:

Reduction or elimination of variable decelerations

Why the other answers are incorrect:

A. Variability does not improve with amnioinfusion.

B. Baseline FHR does not increase as a result of amnioinfusion.

Correct answer: C. Resolution of variable decelerations.

Comprehensive and Detailed Explanation From Exact Extract–Based NCC C-EFM References:

When evaluating an admission tracing, NCC emphasizes determining whether the pattern represents baseline variability abnormalities, signal artifact, or an underlying fetal cardiac rhythm disturbance. The strip shown contains clear features of a fetal dysrhythmia, which NCC and AWHONN describe as an irregular rhythm characterized by inconsistent R–R intervals or intermittent missed beats.

Key features in this tracing:

Extremely irregular FHR signalThe pattern shows abrupt vertical spikes, inconsistent spacing, and intermittent loss of coherent waveform. NCC teaches that this appearance is typical of irregular ventricular conduction or premature atrial/ventricular contractions.

Wide variability in beat spacingBeat intervals vary significantly, suggesting ectopic beats or conduction abnormalities rather than a stable rhythm such as heart block or atrial flutter.

Sensor not malfunctioningThe lower uterine activity channel is smooth and consistent, meaning the upper channel’s abrupt changes represent true FHR signal irregularity, not artifact.

Why the incorrect answers are ruled out:

A. Atrial flutter – NOT supported

Atrial flutter produces a very fast, regular atrial rate (typically 300 bpm) with a repetitive saw-tooth pattern.

It does not produce the highly irregular beat-to-beat pattern seen here.

FHR in atrial flutter appears more organized, not chaotic.

B. Complete heart block – NOT supported

Complete heart block (third-degree AV block) produces a very slow, regular ventricular rate, commonly 50–70 bpm, with a dissociation between atrial and ventricular rhythms.

The tracing here does not show a slow, steady baseline.

Instead, the rhythm is highly irregular with spikes and losses—not characteristic of AV block.

C. Fetal dysrhythmia – CORRECT

NCC, AWHONN, Miller, and Menihan describe fetal dysrhythmias as:“Irregular, inconsistent FHR patterns due to premature atrial contractions (PACs), premature ventricular contractions (PVCs), or intermittent conduction disturbances.”

The hallmark is an irregular rhythm, often appearing as abrupt spikes or missing beats on the monitor.

The tracing shown matches these characteristics precisely.

Therefore, the tracing is most consistent with fetal dysrhythmia, typically benign PACs/PVCs, and is the correct answer.

Comprehensive and Detailed Explanation From Exact Extract (No URLs):

According to AWHONN’s Fetal Assessment Text, Simpson & Miller, and Menihan, vibroacoustic stimulation is utilized during NSTs to elicit fetal accelerations, thereby minimizing testing time.

NCC-referenced sources describe VAS as:

• A method that awakens the fetus,

• Stimulates the fetal auditory system,

• Produces reactive accelerations in a neurologically intact fetus,

• Dramatically shortens NST duration, especially when the fetus is in a sleep cycle.

VAS does NOT measure amniotic fluid, nor does it have any effect on uterine activity (therefore cannot treat tachysystole).

The only correct purpose supported by NCC-cited literature is that VAS shortens the duration of the NST, making Option C correct.